such as CYP7A1 (Ref. 63). It must be determined whether THR analogues protect against, or even reverse, atherosclerosis, and there are limited data in this respect. However, results presented at a ...

该病的发病的主线逻辑，与肝内脂肪细胞死亡以及肝脏细胞纤维化密切相关。一般而言，中年人随着体重的增加，将会导致肝脏内脂肪细胞的集聚，而肝内脂肪细胞的死亡和功能异常一方面将会导致体内胰岛素代谢紊乱，血糖升高，另一方面将会使得HSC（肝脏星型细胞）发生激活，进而促进导致肝脏纤维化。而肝脏纤维化在此前被看做是不可逆的过程。

The proposed mechanisms of action of some of the nutraceutical supplements on the mammalian cholesterol pathway are shown in Figure 2. Virtually all studies have shown very high safety profiles ...

Johanna Honkalammi Mechanism-based inhibition of CYP2C8 by gemfibrozil in humans: characterisation of time and dose relationships. Xiaoqiang Xiang Effects of gender, and SLCO1B1 and CYP7A1 ...